we want to know about a disease, we must first know its pathological changes, because we are not the main aspects of its treatment of clinical manifestations, but the transfer from the sweep of its pathology. In other words we need to fundamentally eliminate the disease. In terms of its pathology Pok pathological changes should be done, because the role of proinflammatory cytokines, an increase in the inflammatory exudate and mucus secretion, epithelial cell degeneration and necrosis that can be severe squamous metaplasia. Furthermore, the respiratory changes in the gland. Because the trachea, bronchial submucosal mucous gland hyperplasia and hypertrophy resulting in a lot of mucus hypersecretion, and even small bronchiole formation of mucus plugs. With the migration of disease, hypersecretion of cell failure, acinar atrophy, mucosal thinning, epithelial squamous metaplasia may be extensive, mucus significantly reduced, there are more lymphocytes tube to avoid plasma cells and other inflammatory cell infiltration. Other pathological changes in elastic fibers and smooth muscle damage to cartilage, weakening the supportive wall, the long wall to form a broad scar. When acute inflammation, when mucosal layer congestion, edema and neutrophil and eosinophil infiltration. There are a lot of pus in the bronchi. The situation is common pathological changes of chronic bronchitis of old, that is, changes in cell Pok.
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Following us from the lung tissue of the dynamic anatomy of curb to know about. When we breathe first thing to do is inhale the gas into the lung tissue, lung capacity increases. Thoracic muscle expansion. When the breath when the gas is discharged from the lungs, thoracic muscle contraction. It is breathing, the respiratory muscles of the role, but when our chest inflammation or trauma cases, patients also experienced chest tightness, shortness of breath performance. This is mainly because we can not breathe when normal chest expansion and contraction. So that gas can not be fully exchanged. Because some patients have phlegm in the trachea when the result can not cough, phlegm blocking the airway, in a slightly longer time when the bacteria invade and easy to form obstructive bronchitis. When we can not rule out thoracic disease case, the passage of time will be the formation of chronic bronchitis. This may be one reason why the old form of chronic bronchitis. As we all know that other factors such as: physical and chemical factors: long-term smoking and inhalation of irritant smoke and dust can damage the respiratory mucosa, the glandular secretion of alveolar macrophages decreased disease resistance. The other cold, cold, temperatures plunged allows bronchial vasoconstriction, decreased ciliary movement and tracheal epithelial filtration and purification reduced. Both infection factors: viruses, bacteria and other infections are a major cause of chronic bronchitis. Allergic factors: This mainly refers to the asthmatic patients. Long-term effects of these factors make the delayed healing and recurrent disease, the pathological basis. Autonomic disorders and adrenal cortex hormones, hormone secretion is too small prostate is one of the causes of this disease.
Since we all know these reasons, it would go above the clinical treatment. Anti-inflammatory, asthma, allergy, enhance physical fitness. But one thing is that we must pay attention to, that is, thoracic and lung tissue of the nerve distribution. Because the body of every organization, every organ functions are coordinated by the nerve function to complete. Whatever the reason for the abnormal nerve conduction will be dominated by its organs and tissues of the functional changes. Loss of function of the original state of equilibrium, so to understand and adjust their disposal nerves, to restore the original function is the key to the treatment of chronic bronchitis. Let's look at that chest and lung tissue of nerve distribution. First introduce the thoracic respiratory muscles, mainly including shoulder and back muscles, muscles on the post, post under the muscle, size, round muscle, put the shoulder muscle, the size of rhomboid muscle, latissimus dorsi, upper and lower serratus anterior, trapezius, and intercostal muscle the back side of the muscle, in front of the pectoralis major, pectoralis minor muscle serratus anterior muscle and so on. Their nerves are: post on the muscle, muscle from the post under the C5, 6 nerve, the size of circular muscle from the C5, 6, C6, 7 nerve, the trapezius muscle by the nerve of the lateral branches, C2-4 nerve , the size of rhomboid muscle from the C2-6 dorsal scapular nerve. So in cervical lesions and involving the same muscle when the patient can feel chest tightness, shortness of breath, chronic bronchitis performance. Other organizations such as the intercostal muscle, chest muscle size and muscle innervation before and after the saw is dominated by the corresponding thoracic nerve. The pleura is innervated by the spinal nerve anterior branch dominant. Innervation of the lung and trachea of the most complex, but also we need to place the focus of discussion. The first is the right vagus nerve behind the line to the right main bronchus down to the back door right lung. Posterior branch of the lung in this issue, and right next to the first 2-5 thoracic sympathetic ganglia consistent with the formation of a branch of the right lung after the plexus. The left vagus nerve in the rear doors separate the multiple branches of the left lung, the first 2-4 and the left paravertebral sympathetic ganglia together form a branch of the left lung after the bundle. That is the movement of the lung and trachea and glandular secretion by the vagus nerve and thoracic spinal cord sympathetic issued jointly to complete. So if in the course of the nerve to walk in, no matter what nerves which part of the emergence of Jire or entrapment, will lead to increased secretion of the gland or trachea and bronchi of the tension caused by excessive weakness or difficulty in breathing spasms. But even so, every nerve of the division of labor is not the same, and is very clear. Vagus nerve in this role is primarily responsible for glandular secretion and metabolism, and sympathetic nerve is responsible for the movement of lung tissue. When the vagus nerve to excessive secretion of glandular tissue can be caused when the blockage occurs moist rales, but when sympathetic stimulation is a time of tracheal and bronchial spasms are often a wheeze, which is We often say that the dry rales. However, the majority of patients with chronic bronchitis are the two rales co-exist. That two types of nerve irritation simultaneously. Sympathetic because we are talking about after the first branch and the branch is connected, so in our examination of such patients tend to occur when the respiratory muscle spasm and pain, muscle cramps when breathing when the lungs will limit organization's campaign to make excretion of tracheal secretions is not smooth worse. Also easy to squeeze next to the spinal nerve in the spine so excited to further increase the contraction of the trachea and bronchi. So when the time of such disease in the treatment of an attack on the more difficult. Similarly, when patients with cervical lesions when the lesions appear to vertebral artery compression, the brain's blood supply will be reduced so that the blood supply to the vagus nerve nucleus and nutrition affected. Vagus nerve in the course of a cranial shift due to cervical vagus nerve can also cause traction. Or cervical nerve appears to control the extrusion of the respiratory muscles cramps, the situation can make the appearance of symptoms of chronic bronchitis.
muscles, improve blood supply to the brain so that normal autonomic nuclear Ruyang, nerve conduction normal activation of the nerve lesion did not shoot, to speed up the absorption and metabolism of inflammatory tissue. treating the symptoms of patients out of pain fast.
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